Cookie preferences

Mandatory cookies are necessary for the basic functionality of a website and cannot be disabled. These cookies are essential for tasks such as allowing users to navigate the website and access secure areas. Mandatory cookies do not collect personal information and are typically set by the website itself. Through these cookies, users can fully experience and interact with the website's essential functions.

Analytical cookies track browsing behaviour over a certain period. Data collected by these cookies are anonymous and for statistical purposes only, such as analysing and reporting visitor interactions with a website. This allows us to further optimize our website.

Functional cookies are vital for optimizing our website functionality. These cookies remember preferences, login details, and language settings. They provide a personalized browsing experience without collecting personal information.

Fowl cholera poultry

Pathology description

Fowl cholera, caused by infection with Pasteurella multocida (PM), is a disease of many avian species. Chickens, turkeys, ducks, and quail are the most important domestic avian species involved and the disease is of economical significance. Pasteurella multocida is a small (less than 2 mm long), Gram negative, nonmotile rod or coccobacillus that varies in virulence depending on the strain. Although PM may induce lesions in multiple organ systems, respiratory pathology is the most important facet of the disease. Pasteurella multocida is capable of multiplication in the bloodstream of a bird. As a result of this bacteremia, the organism may quickly colonize many organs, contributing to the typical purulent exudative lesions of fowl cholera seen in the joints, wattles, ovaries, brain, liver, spleen, and lungs. Shortly before a bird succumbs to the disease, PM will typically multiply to very high levels in the bloodstream and tissues. The incubation period is usually 5-8 days. The bacterium is easily destroyed by environmental factors and disinfectants, but may persist for prolonged periods in soil. Reservoirs of infection may be present in other species such as rodents, cats, and possibly pigs. Predisposing factors include high density and concurrent infections such as respiratory viruses.


• Dejection

• Ruffled feathers

• Loss of appetite

• Diarrhoea

• Coughing

• Nasal, ocular and oral discharge

• Swollen and cyanotic wattles and face

• Sudden death

• Swollen joints (see picture)

• Lameness


Post-mortem lesions

• Sometimes none, or limited to haemorrhages at few sites.

• Enteritis.

• Yolk peritonitis.

• Focal hepatitis.

• Purulent pneumonia (especially turkeys).

• Cellulitis of face and wattles.

• Purulent arthritis.

• Lungs with a consolidated pink ‘cooked’ appearance in turkeys.

Costs of the disease

• Increased feed cost

• Mortality

• Less weight gain